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Moreover, it caused nuclear p53 exclusion and induced apoptosis selectively in senescent Leydig cells by disrupting the FOXO4-p53 interaction, which may be a critical mechanism underlying its therapeutic effect

Kinetics of uptake and deacetylation of N-acetylcysteine by human erythrocytes

& Matsui, H

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This risk association was used in our study to evaluate the relationship between hospitalizations and diseases that are either a cause or a consequence of vitamin B 12 deficiency

The compound is identified by CAS number 307297-39-8, ensuring precise chemical identification and traceability across laboratory documentation, scientific publications, and procurement systems