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Early-stage research suggests that luteolin may elevate the levels of brain-derived neurotrophic factor (BDNF) and amplify the downstream tropomyosin receptor kinase B signaling, which is instrumental in nurturing dendritic spine upkeep, promoting synaptic protein expression, and facilitating processes tied to long-term potentiation ( 3.4 Anti-apoptotic and anti-ferroptotic effects Luteolin exerts neuroprotective effects against programmed cell death by modulating both intrinsic and extrinsic pathways

In some studies it showed therapeutic effects on AD progression [168], but in other studies no effect of idebenone on cognitive decline in AD [169] or on biomarkers related to A and tau pathology in AD was demonstrated [135]

10.12659/MSMBR.883828 [DOI] [PMC free article] [PubMed] [Google Scholar] Jee J

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